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|Title:||Case report: lupus nephritis with autoantibodies to complement alternative pathway proteins and C3 gene mutation.|
Complement Factor B
|Citation:||BMC Nephrol.2015 Mar;(16):40|
|Abstract:||Glomerulonephritis is one of the most severe complications of lupus, a systemic disease with multi-organ involvement, with tissue damage produced mainly by complement activation. As a result of this activation, patients with active lupus present hypocomplementemia during disease flares, but C3 and C4 levels are recovered between episodes.|
|Appears in Collections:||Fundaciones e Institutos de Investigación > IIS H. U. La Paz > Artículos|
Hospitales > H. U. La Paz > Artículos
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