Please use this identifier to cite or link to this item: https://hdl.handle.net/20.500.12530/24540
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dc.contributor.authorMartinez-Martinez, Ernesto
dc.contributor.authorIbarrola, Jaime
dc.contributor.authorCalvier, Laurent
dc.contributor.authorFernandez-Celis, Amaya
dc.contributor.authorLeroy, Celine
dc.contributor.authorCachofeiro, Victoria
dc.contributor.authorRossignol, Patrick
dc.contributor.authorLopez-Andres, Natalia
dc.date.accessioned2019-06-28T14:15:21Z-
dc.date.available2019-06-28T14:15:21Z-
dc.date.issued2016
dc.identifier.citationPLoS ONE.2016;(11)11:e0166272
dc.identifier.urihttps://hdl.handle.net/20.500.12530/24540-
dc.description.abstractGalectin-3 (Gal-3), a β-galactoside-binding lectin, is increased in kidney injury and its pharmacological blockade reduces renal damage in acute kidney injury, hyperaldosteronism or hypertensive nephropathy. We herein investigated the effects of pharmacological Gal-3 inhibition by modified citrus pectin (MCP) in early renal damage associated with obesity and aortic stenosis (AS).
dc.language.isoeng
dc.rightsopenAccess-
dc.subject.meshAcute Kidney Injury
dc.subject.meshAnimals
dc.subject.meshAortic Valve Stenosis
dc.subject.meshDiet, High-Fat
dc.subject.meshDisease Models, Animal
dc.subject.meshFibrosis
dc.subject.meshGalectin 3
dc.subject.meshKidney
dc.subject.meshMale
dc.subject.meshObesity
dc.subject.meshPectins
dc.subject.meshRats
dc.subject.meshRats, Wistar
dc.subject.meshReal-Time Polymerase Chain Reaction
dc.titleGalectin-3 Blockade Reduces Renal Fibrosis in Two Normotensive Experimental Models of Renal Damage.
dc.typeArtículo
dc.identifier.pubmedID27829066
dc.format.volume11
dc.format.pagee0166272
dc.identifier.e-issn1932-6203
dc.identifier.journalPloS one
dc.identifier.doi10.1371/journal.pone.0166272
dc.format.number11
dc.identifier.pmcPMC5102450
dc.pubmedtypeJournal Article
Appears in Collections:Fundaciones e Institutos de Investigación > IIS H. General U. Gregorio Marañón > Artículos

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