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https://hdl.handle.net/20.500.12530/38133| Title: | Benefits of VCE-003.2, a cannabigerol quinone derivative, against inflammation-driven neuronal deterioration in experimental Parkinson's disease: possible involvement of different binding sites at the PPARγ receptor. | |
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| Issue Date: | 16-Jan-2018 | |
| Citation: | J Neuroinflammation.2018 Jan;(15)1:19 | |
| Abstract: | Neuroprotection with cannabinoids in Parkinson's disease (PD) has been afforded predominantly with antioxidant or anti-inflammatory cannabinoids. In the present study, we investigated the anti-inflammatory and neuroprotective properties of VCE-003.2, a quinone derivative of the non-psychotrophic phytocannabinoid cannabigerol (CBG), which may derive its activity at the peroxisome proliferator-activated receptor-γ (PPARγ). The compound is also an antioxidant. | |
| PMID: | 29338785 | |
| URI: | https://hdl.handle.net/20.500.12530/38133 | |
| Rights: | openAccess | |
| Appears in Collections: | Hospitales > H. U. Fundación Alcorcón > Artículos Fundaciones e Institutos de Investigación > IIS H. U. Ramón y Cajal > Artículos | |
Files in This Item:
| File | Description | Size | Format | |
|---|---|---|---|---|
| PMC5771072.pdf | 3.1 MB | Adobe PDF |  View/Open |
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