Please use this identifier to cite or link to this item: https://hdl.handle.net/20.500.12530/41764
Title: Mitochondrial H+-ATP synthase in human skeletal muscle: contribution to dyslipidaemia and insulin resistance.
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Issue Date: 2017
Citation: Diabetologia.2017 10;(60)10:2052-2065
Abstract: Mitochondria are important regulators of the metabolic phenotype in type 2 diabetes. A key factor in mitochondrial physiology is the H+-ATP synthase. The expression and activity of its physiological inhibitor, ATPase inhibitory factor 1 (IF1), controls tissue homeostasis, metabolic reprogramming and signalling. We aimed to characterise the putative role of IF1 in mediating skeletal muscle metabolism in obesity and diabetes.
PMID: 28770317
URI: https://hdl.handle.net/20.500.12530/41764
Rights: openAccess
Appears in Collections:Fundaciones e Institutos de Investigación > IIS H. U. 12 de Octubre > Artículos

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